Touch Football Australia confirms the 2019 National Touch League (NTL) dates and location.Dates: Wednesday 13th March 2019 to Saturday 16 March 2019Location: C.ex Stadium, Coffs Harbour#NTL2019
Touch Football Australia confirms the 2019 National Touch League (NTL) dates and location.Dates: Wednesday 13th March 2019 to Saturday 16 March 2019Location: C.ex Stadium, Coffs Harbour#NTL2019
Entering Saturday’s contest against conference foe Michigan State, the Ohio State men’s basketball team was one of the premiere shooting teams in the country. The Buckeyes came into the matchup ranked 10th in Division I, shooting 49.1 percent from the field. There was no such luck Saturday for OSU at the Schottenstein Center, who lost to the Spartans, 58-48. The loss snapped OSU’s 39-game home winning streak, which was the second-longest active streak in the nation. The Buckeyes shot 26 percent from the field on 14-of-53 shooting, its lowest output since converting 24.6 percent of its shots in a 2007 matchup against Texas A&M in Madison Square Garden in New York, N.Y. Sophomore forward Jared Sullinger struggled all game long, notching 17 points on 5-of-15 shooting. He pulled in 16 rebounds but also committed 10 turnovers. “We were out of sync; we weren’t going to play our offense,” Sullinger said. “Everybody wasn’t paying attention to the plays and didn’t run plays correctly. I think that’s what messed us up the most with our shooting.” It wasn’t only Sullinger who struggled. Streaky senior guard William Buford mustered four points on 2-of-12 shooting from the field. Sophomore forward Deshaun Thomas also only converted 2-of-12 from the field. So why weren’t shots falling for OSU? The Buckeyes had plenty of good looks, and in some cases, it seemed as if they simply couldn’t buy a basket — except from the free-throw line where OSU converted 18 of 22 free throws. “You have to make a shot, have to put the ball in the basket,” OSU coach Thad Matta said after the game. “Some of the looks we had down the stretch were rough. I’m not taking anything away from Michigan State — they had a lot to do with it. I thought our defense was good enough, but you can’t shoot (26) percent.” On first glance, OSU’s offensive output (or lack thereof) was reminiscent of its performance against Kentucky in the 2011 NCAA Tournament, when the Wildcats sent the No. 1-overall seed Buckeyes packing in the Sweet 16, handing OSU a 62-60 loss. The Buckeyes only managed to shoot 32.8 percent from the field in their disappointing season-ender, highlighted by Buford’s dreadful 2-of-16 mark from the field. Michigan State coach Tom Izzo said the play of Buford Saturday, as well as the play from the rest of the OSU squad, was uncharacteristic, but credited it to his defense. “I thought Ohio State missed some shots that I normally see them make,” Izzo said. “We had to run them to wear them down a bit. It was physical both ways.” That it was. The teams combined to shoot 38 free throws in a typically brutal Big Ten slugfest. The slower pace resulted in OSU only producing 48 points, from its average of 76.9 points per game. Sullinger was visibly frustrated at times. He drew plenty of contact in the lane and frequently conversed with officials for foul calls. MSU’s defense collapsed on Sullinger, helping to force his 10 turnovers. “Most of the turnovers I had were me going up for shots,” Sullinger said. “I wasn’t expecting the double (teams) because that’s not what Michigan State shows on film.” Craft said the team needs to regroup. “We have to refocus,” Craft said. “We’re still at the top of the league. We’ve got a long way to go.” Despite handing OSU possibly its ugliest loss of the season, Izzo said he is still impressed by the Buckeyes. “I don’t feel any different than I did a month ago or six months ago,” Izzo said. “Ohio State’s the best team in the league and (Saturday’s game) doesn’t change anything.” read more
The Ohio State football team has granted sophomore defensive back Najee Murray his release from the team, an OSU spokesman confirmed Thursday.OSU coach Urban Meyer said Saturday that Murray had been suspended from the team due to a “training camp issue.” An OSU spokesman confirmed to The Lantern Aug. 7 that Murray had not been practicing with the team.Murray played six games in 2012, primarily on special teams, before suffering a torn ACL in practice. The Steubenville, Ohio native recorded three tackles during his lone season with the Buckeyes.Following Murray’s release, the Buckeyes have 21 players listed as either cornerbacks, defensive backs or safeties on their roster.
Legislation sponsored by state Rep Klint Kesto was signed into law by Gov. Rick Snyder Wednesday, continuing a trend that has made roads safer for Michigan drivers.The legislation maintains the current blood alcohol concentration (BAC) standard at 0.08 through 2021. The decision also ensures the state meets federal guidelines for BAC, allowing the state to qualify for additional road funding.“Our roads are a priority in Michigan,” said Kesto, who was named 2017 Michigan Legislator of the Year by Mothers Against Drunk Driving on Nov. 1. “Maintaining the safety of our residents on Michigan roads is critical, so we must continue the 0.08 BAC benchmark that has made our roads progressively safer over the past 14 years.”In 2000, federal legislation passed requiring all states to establish a 0.08 BAC level for a drunk driving offense by October 2003. Failure to do so would have resulted in federal highway funding sanctions implemented for each year a state failed to pass such legislation.In 2003, Michigan enacted Public Act 61, which lowered the BAC level to 0.08 with a 10-year sunset provision. Public Act 23 of 2013 extended the sunset on the 0.08 BAC level for another five years to Oct. 1, 2018.House Bill 4548 eliminates the Oct. 1, 2018 sunset of the 0.08 BAC standard, setting a new expiration date for Oct. 1, 2021. House Bill 4547, sponsored by Kesto, updates state sentencing guidelines to reflect the changes made in HB 4548.Kesto’s legislation, now Public Act 152 of 2017, was approved by the House and Senate by a combined 144-0 vote.“If we allow the BAC standards to slide backwards, we’ll have people getting the wrong message on drinking then driving,” said Kesto, chair of the House Law and Justice Committee. “This legislation keeps all drivers and their families safe on the road, while also maintaining consistent funding to continue our mission to improve major highways across Michigan.”##### Categories: Kesto News,News 09Nov Rep. Kesto bill ensuring safe roads signed by governor read more
Triax has used its first appearance at the IP&TV World Forum to debut its new IP headend aimed at small operators, MDU installations and the hospitality sector.Hans Lauritsen, export director of Triax, said that small operators were now migrating to IP for the delivery of services, but were nervous about the transition. He said this market segment looked for a single supplier of technology, meaning that it was important to team up with other technology providers to deliver a fully integrated system encompassing elements including middleware and set-top boxes as well as the headend.
Bouygues Telecom CEO Olivier RoussatFrench service provider Bouygues Telecom is to launch its new Android-based box, dubbed Miami, in the New Year, according to local reports.Existing subscribers will be given access to the device in January, with new customers to follow soon afterwards. CEO Olivier Roussat said the operator wanted to reward existing clients for their loyalty first.The box will provide access to Google Play services, as well as services from the likes of Netflix. Although the box is powered by Android, the UI has been designed for Bouygues by French company iFeelSmart, while the recommendation engine is provided by Spideo.The box will be sold at €25.99 a month, initially for ADSL customers, with a fibre version to follow soon after. Bouygues Telecom customers who access services via the Numericable cable network will not, however, have access to the device immediately. read more
Advertisement If you thought the Infinix Hot4 design is good, wait till you get our hands on the Infinix NOte3 Phablet, which was launched unto the Ugandan market through a partnership with Kilimall.DESIGNThis Phablet comes with a 6-inch display, making it a really a big smartphone. It is a slim device with metallic body which gives it a premium look. – Advertisement – The dimensions of the Infinix Note 3 are 161.5 x 81.5 x 8.4 mm and the weight of the smart phone is 200 grams which makes it heavy difficult to use by single hand.At the front display there is LED notification light at the top of the screen on right side that blinks on different notifications, a front facing camera on the left side of the mobile and LED flash light on right side.The earpiece is between the front camera and LED flash light, and the function keys are inside the display screen. At the back, there is a rear camera and dual LED flash below the camera. There is a fingerprint scanner located below the flash.DISPLAYThe phone comes with a 6-inch 1080 x 1920 pixels (480 dpi) display. This gives you the ability to watch your all your high definition content.CAMERAThere is 13 MP, autofocus, dual-LED flash back camera and 5MP front camera.The back camera also supports Gesture Shot, Smile Shot, Panorama.It has different modes, including Picture in picture (PIP) mode which enables you to capture images with both front and rear camera on single screen and merge it.The back camera can record video with 1080 resolution.If you are into photography, the Note3 will come in handy.SOFTWARE AND SPECIFICATIONSThe Note3 runs on Android 6 Marshmallow under Infinix Mobility’s XOS user interface.It comes with a number of apps pre-installed, which include: Ultra Power, XManager, XAccount, XClub, XHide and XWeather.BODYDimensions 161.5 x 81.5 x 8.4 mmWeight 200 gSIM Slot Dual SIM (Micro-SIM, dual stand-by)DISPLAYType FHD IPS capacitive touchscreenSize 6.0 inches (~75.3% screen-to-body ratio)Resolution 1080 x 1920 pixels (480 dpi)Multi-touch Yes, 5-PointNETWORKNetwork Support GSM/3G2G Bands 900/18003G Bands 2100Speed HSPA 42.2/5.76 MbpsPLATFORMOs Android OS v6.0 (Marshmallow), XOS ChameleonChipset Media Tek MT6753CPU Octa-core 1.3 GHz, ARM Cortex-A53, 64-bitGPU Mali-T720MEMORYCard Slot Yes, up to 128GBinternal 16 GB, 2GBCAMERAPrimary 13 MP, autofocus, dual-LED flashFeatures Gesture Shot, Smile Shot, Panorama Video 1080p @30fps Secondary 5 MPCOMMSWLAN Wi-Fi 802.11 b/g/n, hotspotBluetooth v4.0GPS Yes, with A-GPSRadio YesUSB v2.0, microUSBFEATURESSensors Fingerprint, accelerometer, gyro, proximityBATTERY Non-removable Li-ion 4500 mAh battery BATTERYThere is a Li-ion 4500 mAh non-removable battery is fixed in Infinix Note 3. This is powerful compared to other phablets. The Note3 also has standby intelligent power saving mode and supports fast charging.In Ultra power mode, all power functions are tunrned off to extend the using time.PRICEThe Infinix Note3 is currently selling at only UGX 519,000 at kilimall.co.ugHigh points.Great battery.Finger print sensorGood Camerasupports Fast chargeDual SIMLow points.No 4G LTE(Which is now a standard)Battery is not removable, not easily replaceable.Heats up when Camera is used for long. read more
Advertisement TECNO Mobile has announced the #GivingBackWithTECNO corporate social responsibility (CSR) drive. That will see the mobile phone company join hands with its customers during the Holy month of Ramadan and give back to society, in particular Sanyu Babies Home on Eid Day.The CSR drive commenced on the 5th June and will end on 24th June, involving different activities across the city that will see customers buy selected TECNO Mobile models that is; the CX, CX Air, L9, L9 Plus, Phantom 6 and the Phantom 6 Plus from participating dealership shops in Kampala. A portion of the money from the sold models will be directed towards the #GivingBackWithTECNO campaign and will finalize with a visit to Sanyu Babies Home with selected items to donate to Children on Eid day.The #GivingBackWithTECNO CSR drive will take a period of 3 weeks over which TECNO Mobile invites Ugandans to join it, in giving back by purchasing the selected models above. Amongst these models are TECNO’s latest Camon CX and CX Air, both best known for taking quality pictures, the L9 and L9 Plus also known for having outstanding batteries that can last over 72 hours without charge. – Advertisement – A portion of the proceeds from the sale of these smartphones will go towards buying items for Sanyu Babies Home and these will be delivered on Eid day on top of the company’s own contribution. The activities will run parallel both in the selected TECNO partner shops and on TECNO’s social media channels. Participating customers will also be awarded with assorted TECNO gifts as appreciation for contributing towards this cause.Last year, TECNO visited the Ntinda School of the Deaf in a similar CSR drive and pledges to continue giving back to Ugandan society in the upcoming years.Speaking during the launch of the #GivingBackWithTECNO campaign, TECNO’s marketing manager, Mr. Ralap Yang commented, “TECNO Mobile Uganda is doing this campaign during Ramadan in the hope that we can work together with our mobile phone users as well as potential users and help people in need. We thought of children in the orphanages and care homes who need love and care. We stayed true to the meaning of Ramadan through kindness and we believe Allah will appreciate it. I also hope that in the future, we can have more loyal TECNO fans join us and we work together in doing more kind deeds. “[related-posts] read more
Patients who suffer from Restless legs syndrome complain of painful symptoms in the legs leading to sleep disturbances. The mechanisms for RLS are still not completely understood. We have shown that also the nerve cells supplying muscles in the leg are responsible and hereby additional drug treatments may be ahead targeting these nerve cells.”Dirk Czesnik, Co-author Nov 13 2018Reviewed by Kate Anderton, B.Sc. (Editor)New research published in The Journal of Physiology presents a breakthrough in the treatment of Restless Legs Syndrome (RLS).RLS is a common condition of the nervous system that causes an overwhelming irresistible urge to move the legs. Patients complain of unpleasant symptoms such as tingling, burning and painful cramping sensations in the leg. More than 80% of people with RLS experience their legs jerking or twitching uncontrollably, usually at night.Until now it was thought that RLS is caused by genetic, metabolic and central nervous system mechanisms. For the first time the researchers show that, in fact, it is not only the central nervous system but also the nerve cells targeting the muscles themselves that are responsible.Related StoriesStudy finds high rates of small intestinal bacterial overgrowth in patients with restless legs syndromeOlympus Europe and Cytosurge join hands to accelerate drug development, single cell researchScientists develop universal FACS-based approach to heterogenous cell sorting, propelling organoid researchThis new research indicates that the involuntary leg movements in RLS are caused by increased excitability of the nerve cells that supply the muscles in the leg, which results in an increased number of signals being sent between nerve cells. Targeting the way messages are sent between nerve cells to reduce the number of messages to normal levels may help prevent the symptoms of RLS occurring. This could be achieved by new drugs that block the ion channels that are essential for the communication between nerve cells.The research conducted by the University of Gottingen in conjunction with the University of Sydney and Vanderbilt University involved measuring the nerve excitability of motor nerve cells of patients suffering with RLS and healthy subjects.The next step is to investigate the effect of different medications in patients and the effect on RLS. Source:http://www.physoc.org/ read more
Reviewed by James Ives, M.Psych. (Editor)Feb 12 2019Biologist Aleksey Belikov from the Moscow Institute of Physics and Technology has proposed that rapid progression of age-related diseases may result from the formation of so-called vicious cycles. An example of this is when toxic products of a biochemical reaction trigger that same reaction to happen again. The study, published in the January issue of Ageing Research Reviews, highlights the most promising options for age-related disease treatment that focus on interrupting vicious cycles.Statistical data indicate that the mortality rates due to all major age-related diseases increase exponentially with age. The MIPT researcher hypothesized that the reason behind this self-aggravating disease progression is the indefinite repetition of reaction cycles, which increases the harm from the initially noncritical changes in the body manyfold. It is these cycles that prospective therapies might address.”Age-related diseases in humans are nearly impossible to prevent, though the search for so-called geroprotectors continues,” said the paper’s author Aleksey Belikov, who holds a doctorate in natural sciences and is a researcher at MIPT’s Laboratory of Innovative Medicine. “The therapies that are being developed now aim to prevent disease progression.””Investigating the mechanisms behind age-related disease progression, one concludes that by the time the disease has been diagnosed, it is too late to address the triggering factors,” the biologist explained. “Apparently the most effective strategy is to interrupt the known vicious cycles by blocking certain stages in them. Drugs doing just that are already being developed.”In his study, Belikov examined the mortality rates of patients with five most widespread diseases that tend to affect elderly people more often, leading these diseases to be widely regarded as age-related: atherosclerosis, hypertension, diabetes, Alzheimer’s and Parkinson’s diseases. Mortality statistics are the most powerful and least biased tool for studying diseases, since they account for the natural progression of a disease under various life conditions across a large population.Exponential, or geometric, growth is observed in nuclear reactions, water freezing, bacterial growth, avalanches, and other phenomena. To use the example of bacteria, one might imagine a bacterium in a test tube that divides once every second, and its offspring divide once every second, etc. Say it takes the bacteria four hours to fill up the entire test tube. The tube will be half-full just one second before the end of the experiment, yet four seconds earlier it’s just over 3 percent full. That is, the growth rate accelerates rapidly.Related StoriesStudy provides new insight into longitudinal decline in brain network integrity associated with agingNew methods to recognize antimicrobial resistant bacteria and how they workNew research could help design algae that produces fuels and cleanup chemicalsA detailed analysis of age-related diseases revealed that they progress exponentially due to reactions on the molecular or cellular level producing pathogenic products which in turn initiate the very reactions that produced them. That way the harmful products quickly multiply, and the disease progresses at an ever increasing rate, like an avalanche.The MIPT researcher investigated the mechanisms of each of the five age-related diseases in the study, identifying the molecular and cellular reactions that result in vicious cycles. He noted that even substances or factors which do not in themselves pose any critical health risks might nevertheless initiate a vicious cycle of reactions. As a result, the amount and impact of the cycle’s toxic component is amplified uncontrollably. This means that a lethal effect might stem from an initially noncritical amount of a harmless substance.For example, nerve cells in the brain contain a small amount of a protein called alpha synuclein, which is involved in nerve impulse transmission. It may happen that the gene encoding alpha synuclein is mutated, duplicated, or triplicated in a genome. This leads to multiple protein molecules sticking to one another, forming so-called toxic oligomers, which then grow in size by attaching other alpha synuclein molecules. This process produces fibrils, which from time to time break up into oligomers, each of which eventually grows into a new fibril, etc. This chain reaction causes the number of toxic alpha synuclein oligomers to grow exponentially, forestalling the exponential growth of the mortality rate due to Parkinson’s disease with patient age. While it is almost impossible to eliminate the oligomers that have already formed, their further growth might be prevented pharmacologically, halting the progression of the disease.”All the vicious cycles this paper deals with are based on prior studies of age-related diseases,” said Belikov. “New studies could complement or even radically change our understanding of what reactions constitute the cycles. Yet the notion of positive feedback loops, or vicious cycles, behind the exponential progression of age-related diseases will most likely remain.”? Source:https://mipt.ru/english/news/mipt_biologist_explains_molecular_mechanisms_behind_rapid_progression_of_age_related_diseases read more
Source:https://nyulangone.org/ Reviewed by James Ives, M.Psych. (Editor)May 2 2019The action of a gene called ATDC is required for the development of pancreatic cancer, a new study finds.The work builds on the theory that many cancers arise when adult cells — to resupply cells lost to injury and inflammation — switch back into more “primitive,” high-growth cell types, like those that drive fetal development.When this reversion happens in the presence of other genetic mistakes, a repair process meant to start and stop quickly continues unchecked.New details of this cancer-causing switch to primitive cells, and of the role of ATDC in pancreatic cancer formation, are revealed in a study of mice and human patient samples published online May 2 in the journal Genes & Development.Led by researchers from NYU School of Medicine and the University of Michigan, Ann Arbor, the study found that ATDC must be active if pancreatic cells, when injured, are to re-acquire primitive stem-cell qualities and undergo the early steps that lead to the development of pancreatic cancer.”We found that deleting the ATDC gene in pancreatic cells resulted in one of the most profound blocks of tumor formation ever observed in a well-known mice model engineered to develop pancreatic ductal adenocarcinoma, or PDA, which faithfully mimics the human disease,” says corresponding author Diane Simeone, MD, director of the Pancreatic Cancer Center of NYU Langone Health’s Perlmutter Cancer Center. “We thought the deletion would slow cancer growth, not completely prevent it.”The search for better treatment in these cases is especially urgent, says Simeone, given that PDA has the worst prognosis of any major malignancy and is on track to become the second leading cause of cancer-related death by 2030.Healing Gone AwryThe study focused on acinar cells in the pancreas that secrete digestive enzymes through a network of partnering ducts into the small intestine. These same digestive enzymes can subject this tissue to low levels of damage. In response, acinar cells have evolved to readily switch back into stem cell types resembling their high-growth ancestors, a feature that they share with pancreatic duct cells.This ability to regenerate comes at a price, researchers say, because such cells are prone to become cancerous when they also acquire random DNA changes, including those in the gene KRAS that are known to drive aggressive growth in more than 90 percent of pancreatic cancers.Related StoriesSpecial blood test may predict relapse risk for breast cancer patientsNew gene-editing protocol allows perfect mutation-effect matchingNew study to ease plight of patients with advanced cancerSpecifically, stressed acinar cells are known to temporarily undergo acinar-to-ductal metaplasia or ADM, a step toward a primitive cell type to resupply cells. This sets the stage for a second shift into pancreatic intraepithelial neoplasia (PanIN), in which cells no longer multiply under normal controls.In the current study, the researchers found that mutant KRAS and other genetic abnormalities induced aggressive pancreatic cancer in 100 percent of study mice when the ATDC gene was present, but in none of the same cancer-prone mice lacking the gene. Neither did acinar cells in the ATDC “knock-out” mice undergo ADM or transformation to PanIN.To get a better look at the early steps in pancreatic cancer formation, the research team artificially caused pancreatitis in mice by treating them with cerulein, a signaling protein fragment that damages pancreatic tissue. ATDC gene expression did not go up right after the damage, but increased a few days later and in line with the timeframe required for acinar cells to reprogram genetically into their ductal cell forebears.Further experiments confirmed that the expression of ATDC triggers beta-catenin, a cell-signaling protein that, upon receiving the right trigger, activates genes that include SOX9. Earlier studies linked SOX9 to the development of ductal stem cells and to the aggressive growth seen in PDA. Consistent with this work, the current study found that the inability of cells lacking ATDC to become cancerous was due to their inability to induce SOX9 expression.The authors also examined ATDC expression in ADM lesions from 12 samples of human pancreatic tissue. The team found it to be more active in human ADM lesions along with beta-catenin and SOX9, and its activation increased further during the transition of ADM into human pancreatic ductal adenocarcinoma.The findings, says Simeone, identify ATDC, beta catenin, SOX9, and their signaling partners as potential targets in the design of new treatment approaches and prevention strategies for pancreatic cancer. read more
© 2018 AFP Citation: CBS sues to block effort to ‘force’ merger with Viacom (2018, May 14) retrieved 18 July 2019 from https://phys.org/news/2018-05-cbs-sues-block-effort-merger.html US media giant CBS filed suit Monday, alleging that majority shareholder Shari Redstone is seeking to “force” a merger with rival Viacom on unfavorable terms. Explore further This document is subject to copyright. Apart from any fair dealing for the purpose of private study or research, no part may be reproduced without the written permission. The content is provided for information purposes only. A lawsuit alleges that controlling CBS shareholder Shari Redstone is seeking to force a merger with media rival Viacom on unfavorable terms for CBS Viacom names Robert Bakish acting president, CEO The lawsuit filed in Delaware Chancery Court claims “breaches of fiduciary duty” by Shari Redstone and National Amusements, the holding company created by her father, 94-year-old Sumner Redstone.CBS is seeking a temporary restraining order “to protect the status quo and to prevent imminent and irreparable harm” from merging CBS and Viacom, two media groups which were split a decade ago by Sumner Redstone.The lawsuit alleges that Shari Redstone is seeking “to force through a merger of CBS and Viacom on terms that are contrary to the best interests of the public stockholders—without allowing them any voice on the transaction.”This effort “would subvert the board-approved Special Committee process,” evaluating a tie-up of the two firms.CBS said in February it had established a committee to consider reuniting the two companies, combining CBS with Viacom’s networks which include Nickelodeon, MTV, BET and Comedy Central.The companies previously explored a combination in 2016, but later abandoned the effort.Shari Redstone, acting on behalf of her father, effectively controls both firms through National Amusements through special voting shares.CBS has called a special board meeting for Thursday that will consider issuing a dividend that would dilute the voting power of the holding company from 79 percent to 17 percent.The dividend “would enable the company to operate as an independent, non-controlled company and more fully evaluate strategic alternatives,” a CBS statement said.The lawsuit said Shari Redstone could force a merger by replacing CBS board members.”If Ms. Redstone can replace Board members or modify the Company’s governance documents in the next three days, it is uncertain that the board will be able to protect all stockholders by considering at the next Board meeting whether to take action in response to her threats and breaches of fiduciary duty,” the suit alleges. read more
7-year-old in Mumbai’s Dharavi drowns after falling into drain, 3rd incident in a weekIn the third such incident in Mumbai, a 7-year-old boy fell down an open drain and died on Monday.advertisement Next India Today Web Desk New DelhiJuly 15, 2019UPDATED: July 15, 2019 19:11 IST Family members grieve the boy who died on Monday in Dharavi. (Image: India Today)A seven-year-old boy in Mumbai’s Dharavi area died due to drowning after he fell down a drain at the Rajiv Gandhi Colony. This is the third such incident in a week.After being alerted about the incident, police rushed to spot and pulled up the boy from the open drainage, reported PTI.He was rushed to the Sion hospital, where he was declared brought dead, confirmed an official.This comes even as the body of 1.5-year-old Divyansh, who fell into a gutter in Goegaon on July 10, is yet to be recovered.The infant was strolling outside his house at night when Mumbai was being pounded by heavy rain and he slipped into the open gutter.As the inconsolable family cried for help and protested on the streets, BMC and police personnel swung into action.However, they are yet to find the body and fear that the little boy may have been swayed into the Arabian Sea.Now, just days later, another boy has drowned in a drain in a similar incident.Meanwhile, on Friday, a 12-year-old boy died after falling into a water-filled pit, dug for the construction of the Coastal Road near Worli.Bablu Kumar Paswan drowned in the pit near Worli Sea Link at 1.20 pm on Friday, said an official of the Brihanmumbai Municipal Corporation (BMC).(With PTI inputs)Also Read | Mumbai rains: Locals stage protest against BMC for inaction, failure to trace missing 18-month-old infantAlso Watch | 2-year-old falls into open drain in MumbaiFor the latest World Cup news, live scores and fixtures for World Cup 2019, log on to indiatoday.in/sports. Like us on Facebook or follow us on Twitter for World Cup news, scores and updates.Get real-time alerts and all the news on your phone with the all-new India Today app. Download from Post your comment Do You Like This Story? Awesome! Now share the story Too bad. Tell us what you didn’t like in the comments Posted bySanchari Chatterjee read more
Science Fact or Fantasy? 20 Imaginary Worlds The 18 Biggest Unsolved Mysteries in Physics 5 Mars Myths and Misconceptions There are turbulent, unexpected currents crackling through Jupiter’s atmosphere, producing brilliant auroras. Juno, the NASA probe that has orbited the gas giant since 2016, passes over Jupiter’s polar regions ever 53.5 days, collecting data on the magnetic forces that produce ultrabright auroras above the huge planet. In a new paper, published July 8 in the journal Nature Astronomy, researchers working with Juno’s data discovered that the electric currents passing through Jupiter’s magnetosphere — the region of its atmosphere richest with magnetic field lines — don’t act as expected. The probe found less direct current — current that constantly flows in one direction — than physicists predicted. It was only about 50 million amperes, an incredibly powerful current, but not as high as theoretical models of Jupiter’s magnetosphere suggested would be present. That finding suggests that “alternating current” — current that flickers back and forth — plays a much bigger role in producing Jupiter’s auroras than anyone realized, the researchers wrote. On Jupiter, as on Earth, auroras are a product of whirling currents in magnetic fields interacting with high-energy particles from the sun. [10 Places in the Solar System We’d Most Like to Visit]Headbutting Tiny Worms Are Really, Really LoudThis rapid strike produces a loud ‘pop’ comparable to those made by snapping shrimps, one of the most intense biological sounds measured at sea.Your Recommended PlaylistVolume 0%Press shift question mark to access a list of keyboard shortcutsKeyboard Shortcutsplay/pauseincrease volumedecrease volumeseek forwardsseek backwardstoggle captionstoggle fullscreenmute/unmuteseek to %SPACE↑↓→←cfm0-9接下来播放Why Is It ‘Snowing’ Salt in the Dead Sea?01:53 facebook twitter 发邮件 reddit 链接https://www.livescience.com/65951-jupiter-currents-magnetosphere-tesla.html?jwsource=cl已复制直播00:0000:3500:35 “These observations, combined with other Juno spacecraft measurements, show that alternating currents play a much greater role in generating Jupiter’s aurora than the direct current system,” Joachim Saur, an author of the paper, said in a statement. On Earth, we typically think of alternating and direct currents (AC and DC) in terms of electronics. Famously, in the late 19th century, inventors Thomas Edison and Nikola Tesla disagreed sharply over which method should be used to deliver power to electrical devices. DC power doesn’t convert as easily between different voltages, according to the U.S. Department of Energy (DOE), so Tesla wanted to turn the more-easily convertible AC into the standard. Edison, guarding his DC-dependant patents, resisted the change and spread misinformation that AC was more dangerous, according to the DOE. Tesla won out in the end, and AC became the standard for U.S. power plants. However, according to the DOE, direct current has regained favor as more battery-powered devices have come to market. Your lights are probably running on AC power, but there’s a good chance the device you’re reading this on relies on DC. (That’s why your laptop requires an AC adapter.) In the space around Jupiter, the proportion of AC to DC isn’t determined by feuding pre-modern inventors, but by the behavior of ions in the planet’s atmosphere. Jupiter has powerful currents than Earth for several reasons, including its huge size, its fast rate of spin and the excess of charged particles (ions) pumped out from volcanoes on the moon Io. That such a large proportion of those currents are AC seems to be a result of turbulence in the planet’s magnetic fields, the researchers wrote. Turbulence in this sense refers to the disordered way in which the magnetic fields’ shape and directionality fluctuates. And that turbulence is producing different effects at each of Jupiter’s two poles. In the time Juno has orbited Jupiter, the planet’s north pole has experienced about half the current of the south pole, the researchers wrote. That seems to be a result of the much more complex arrangement of magnetic field lines in the north, which interrupts the flow of currents. In the south, they wrote, the magnetic field lines are “smoother.” The effects of those differences are visible in the two poles’ auroras, they noted. In the north, the auroras tend to be more widely dispersed, with a structure of “filaments and flares.” In the south, the auroras tend to be more structured, with a “bright arc” extending out from the main oval where auroras occur. This research on Jupiter’s powerful magnetic fields, the researchers wrote, could inform their understanding of Earth’s weaker magnetic field — humanity’s main protection against harsh solar particles. Some researchers already suspected turbulence produced a significant proportion of currents around our planet. This work seems to lend credence to that idea. 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COMMENT SHARE The apex court fixes the next hearing on December 5 CBI Director Alok Verma, who has been divested of all duties by the Centre and sent on leave, told the Supreme Court on Thursday that he was appointed for a fixed tenure of two years and cannot even be transferred.Verma, who has a feud with his deputy Rakesh Asthana, Special Director in CBI, had approached the apex court seeking stay on the Centre’s order contending that it was against the guidelines laid down by the top court.Challenging the government’s decision, Verma’s counsel and senior advocate Fali S Nariman said the CBI Director was appointed on February 1, 2017 and “the position of law is that there will be a fixed tenure of two years and this gentleman cannot be even transferred.”Making his submission before the bench, also comprising Justices S K Kaul and K M Joseph, Nariman said there was no basis for the Central Vigilance Commission to pass an order recommending that Verma be sent on leave.“There has to be strict interpretation of the Vineet Narain judgment. This is not the transfer and Verma has been denuded of his power and duties…otherwise there was no use of the Narain judgement and the law,” Nariman said.The Vineet Narain decision, delivered by the apex court in 1997, relates to the investigation of allegations of corruption against high-ranking public officials in India. Before 1997, the tenure of the CBI directors was not fixed and they could be removed by the government in any manner. But the apex court in the Vineet Narain judgment fixed a tenure of a minimum of two years for the CBI director to allow the officer to work with independence.Nariman referred to the terms and conditions of appointment and removal of the CBI Director and concerned provisions of the Delhi Special Police Establishment (DSPE) Act, 1946.Attorney General K K Venugopal opposed the plea of Verma and others that the government should have gone back to the selection panel for divesting the CBI Director of his duties and sending him on leave. He told the court that Verma is in Delhi living in the same house and hence there is nothing to construe that he has been transferred.“Verma continues to hold the status of Director, and enjoys all privileges and benefits of the post till date,” the AG told the bench, which adjourned the hearing for December 5.Senior advocate Kapil Sibal, appearing for Leader of Opposition Mallikarjun Kharge (Congress), referred to the CVC order divesting Verma of his power and sending him on leave. He said the CVC and the government cannot override DSPE provisions which makes it mandatory to consult panel before “transfer” of CBI chief mid-tenure.“The order to seal Verma’s office passed without powers under the CVC Act,” Sibal said. He said that even if a CBI Director is caught red-handed, the government has no choice but go the selection panel. “Remedy of acting against the CBI director lied with the selection committee and I am not saying that no action can be taken against the CBI Director,” Sibal said.“Power to appoint will also have power to suspend or dismiss. This is why whatever had to be done against Verma, it had to go to the Selection Committee. This may tomorrow happen to CAG, CVC since similar provisions are for them,” Sibal said. The senior advocate asked, “What will happen to the independence of CBI if government is allowed to remove the CBI Director like this?” SHARE SHARE EMAIL November 29, 2018 COMMENTS Published on judiciary (system of justice) corruption & bribery read more